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doi: 10.4049/jimmunol.1302286 93 MooreSJuoHHNielsenCTTydenHBengtssonAA

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96 Additionally, AMPK activation by reduced ATP levels maintains NADPH through inhibiting NADPH consumption in fatty-acid synthesis and enhancing NADPH production from fatty-acid oxidation instead of PPP to inhibit cell death

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doi: 10.3390/pathogens13070565

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In recent in vitro assays performed by our group [32], we showed that H 2 O 2 -induced oxidative stress triggered the binding of Prx2 and GPx to RBC membrane

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